The sands of time are falling away. Better get sketching.
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A note regarding June 12-13 and the announced Reddit API changes.
Theme posted by topherthepest Tomorrow: Free Draw Friday

🕉️ experienced growers, kindly share your thoughts on my first cultivation. Lady details - Age: 10 weeks since gerimnation Genetics: grown from country side unbreaded seed Lighting: - 10 hours direct sunlight - 2 hours of indirect sunlight - 12 hours of complete darkness Positioning: placed on very good natural air circulated window, living in 5 gallon pot with a potting mix soil Nutrition: - Cal/Mag once every 2 weeks - Epsom salt once every 4 weeks - Rock phosphorus added only once in 9th week
Images: (Note all images at the left bottom have caption) 1. Is she looking good for a untrained lady? No topping, no LST 2. Started seeing these hair from 10th week. Are those pistils & trichomes over it? I'm seeing those only on 3 joints of stem & side branches (near stipules area), should this abundancy be normal & keep on growing with age? 3. Top view of image shows any signs of pre-flowering/flowering? I'm seeing those white hair starting to grow as fan leaves or am I wrong to understand it? 4. Side view of branch with terminal node? Any signs of pre-flowering? 5. Saw these white spots on lady, after researching got know it's been attacked by spider mites. Immediately removed some of the majorly damaged fan leaves Killed them without any mercy with my bare hands as many I could find them beneath the leaves. Sprayed them with diluted solution of neem oil. Any other measures can be taken?
All from the above notes, can growers provide some relative advice which I should follow!? Trying to learn this art of growing by watching YouTube videos & reading books to understand the process.
Although we're far to reach in the future, but any estimates on weight from the yield?
Need some pat on the back, saying we're on correct track 😁 (pun intended)
Thanks to community!

I know it's been a long time since release, however I didn't get my suit. It was supposed to be given to players who play in the first week after the launch, which was December 12. I first played 5 days after the release, December 17.
Now, I remember there were a lot of issues and bugs with this suit, however I think that it should have been fixed by now. So now what?

I'm trying to optimise a Drow Bard and intending to go the College of Valour route, other than spells I will mainly be using Finesse Weapons and Ranged attacks, so Dex will be 16. Is it viable to dump Str to 8, or will I need to go for Str 10 in order to be able to carry Medium Armour, Shield, Heavy crossbow and a selection of Finesse weapons (Rapier, Scimitar etc.) or is that going to create inventory management problems? If Str 8 is viable, I could go for for something like:
Str 8 Dex 16 Con 14 Int 8 Wis 12 Cha 16
Wisdom is useful for many spell saves and Perception checks (I get Perception skill for free as a Drow) for spotting traps etc. (my Bard will also be my main thief)
If I'm going to need Str 10 though would probably go for:
Str 10 Dex 16 Con 14 Int 8 Wis 10 Cha 16
Also, without giving away spoilers, is Int 8 going to be a problem? I will have other party members with higher Int (e.g. Gale), but don't want to fail important saves too easily.

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Having completed high school before the K-12 program, we were thrust into deciding which career path we are going to take at the age of 15-16. My questions are:
Was it too young for us to decide what our eventual lives would be spent on?
How much of the decision-making was made by our parents and what they wanted instead of what we were passionate about?
Are you happy with your career now? Not content, but happy.

Hi all, firstly I should mention im not a complete novice rider I rode consistently for 12 years plus from the age of 6.
I have had a very long break though and now mid 40s (stopped due to moving, no transport to get to a riding school and having my 15 Yr old son).
I now have transport but the only school close by doesn't do single lessons just groups. Not sure how I'd feel in a group of kids tbh (no offence to kids I'd just feel embarrassed) the woman who owns the school hasn't really been helpful when I asked if they do adult groups and just said 'mixed'
Do I go anyway or look further afield to find a school that does solo lessons?
I'm in the UK btw.

Hi everyone, Today we just had our neckfold scanning, and we got a measurement of 3:1, and therefore a risk for chromosome 21 / downsyndrome of 1:273. They therefore offered us NIPT test, placenta testing or amniocentisis.
We already had an NIPT scan last week and just got the results that they are normal, so it would not make sense to repeat the NIPT. We are considering CVS only because it could be done asap rather than ambicentisit (12 weeks pregnant now), but scared of the risk for miscarriage..
Any experiences with CVS, both good and bad? Is 1:273 a huge risk?
Thank you so so much!

Hello fellows, familia. Grab a coffee/joint/gamersupp.
This is just a public health awareness thread about some little known things amongst the normans. Reddit wont let me post links, but what I will do is post study title and authors/year, and some cases abstract copy paste. I will do at the bottom after the statements.
So, as you all know, COVID is a thing still, and many didn't actually know what it was capable of, outside of comparisons to other coronaviruses and the acute illness. Right now, acute illness is incredibly manageable, but the long term effects on organs is becoming alarming, even in mild cases. So lets take it from the top, so we are on the same page. We will start on organ systems and virus character.
So, covid is pretty much a vascular virus, which seems to disrupt vasculature. Lets start before vaccines to demonstrate to those among us suspicious that the vaccine is the cause. (Its fine to worry about tech, i dont judge).
Every person in Sweden diagnosed with COVID-19 from Feb. 1, 2020, to Sept. 14, 2020 — a total of nearly 87,000 people. Their median age was 48, and 57% were women. Researchers compared them with more than 348,000 Swedish people of similar age and sex who did not have the virus.
In the week after a COVID-19 diagnosis, the risk of a first heart attack increased by three to eight times. The risk of a first stroke caused by a blood clot multiplied by three to six times. In the following weeks, both risks decreased steadily but stayed elevated for at least a month.
Thats PRE vax. In a population which probably have the obesity % of the US and a healthy hiking and outdoor culture. US obesity rate is 40%+. Sweden perhaps half or less. Also, viral load on exposure can be a large determination of severity acutely, but perhaps also viral creep inside the body and the resulting dysfunction and damage.
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Risk of Cardiovascular Events after Covid-19: a double-cohort study Objective To determine absolute and relative risks of either symptomatic or asymptomatic SARS-CoV-2 infection for late cardiovascular events and all-cause mortality.
Conclusions Either symptomatic or asymptomatic SARS-CoV-2 infection is associated with increased risk of late cardiovascular outcomes and has causal effect on all-cause mortality in a late post-COVID-19 period
Characteristics of a COVID-19 Cohort With Large Vessel Occlusion: A Multicenter International Study

CONCLUSION:

COVID-19 was an independent predictor of incomplete revascularization and poor outcomes in patients with stroke due to LVO. Patients with COVID-19 with LVO were younger, had fewer cerebrovascular risk factors, and suffered from higher morbidity/mortality rates.
Long-term cardiac pathology in individuals with mild initial COVID-19 illness
In this cohort of selected previously well and home-isolating individuals initially evaluated at a median of 109 days and followed-up at 329 days after diagnosis of COVID-19 infection, respectively, the ongoing cardiac symptoms were related to mild but notable imaging findings, suggesting inflammatory cardiac involvement. At baseline, participants with cardiac symptoms had higher mapping values, suggesting diffuse myocardial inflammation, and more frequent pericardial enhancement, suggesting pericardial inflammatory involvement compared to asymptomatic participants. At follow-up, 53% of the cohort had persistent cardiac symptoms, whereas new symptoms developed in 5%.
Transcriptomic profiling of cardiac tissues from SARS-CoV-2 patients identifies DNA damage
In this study, we investigated the host transcriptome landscape of cardiac tissues collected at rapid autopsy from seven SARS-CoV-2, two pH1N1, and six control patients using targeted spatial transcriptomics approaches. Although SARS-CoV-2 was not detected in cardiac tissue, host transcriptomics showed upregulation of genes associated with DNA damage and repair, heat shock, and M1-like macrophage infiltration in the cardiac tissues of COVID-19 patients. The DNA damage present in the SARS-CoV-2 patient samples, were further confirmed by γ-H2Ax immunohistochemistry.
In comparison, pH1N1 showed upregulation of interferon-stimulated genes, in particular interferon and complement pathways, when compared with COVID-19 patients. These data demonstrate the emergence of distinct transcriptomic profiles in cardiac tissues of SARS-CoV-2 and pH1N1 influenza infection supporting the need for a greater understanding of the effects on extra-pulmonary organs, including the cardiovascular system of COVID-19 patients, to delineate the immunopathobiology of SARS-CoV-2 infection, and long term impact on health.
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Great article by British Heart Foundation.

Is coronavirus a disease of the blood vessels?

Updated 12 May 2023
Dr Phoebe Kitscha explores how and why Covid-19 affects the whole circulatory system, and the research that is trying to tackle it.
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March 27, 2020

Potential Effects of Coronaviruses on the Cardiovascular SystemA Review

'Coronavirus disease 2019 is associated with a high inflammatory burden that can induce vascular inflammation, myocarditis, and cardiac arrhythmias. Extensive efforts are underway to find specific vaccines and antivirals against SARS-CoV-2. Meanwhile, cardiovascular risk factors and conditions should be judiciously controlled per evidence-based guidelines.'
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2022
Long-term cardiovascular outcomes of COVID-19
our results provide evidence that the risk and 1-year burden of cardiovascular disease in survivors of acute COVID-19 are substantial. Care pathways of those surviving the acute episode of COVID-19 should include attention to cardiovascular health and disease.
Acute and postacute sequelae associated with SARS-CoV-2 reinfection
The evidence shows that reinfection further increases risks of death, hospitalization and sequelae in multiple organ systems in the acute and postacute phase. Reducing overall burden of death and disease due to SARS-CoV-2 will require strategies for reinfection prevention.
2020
Long covid: Damage to multiple organs presents in young, low risk patients
Initial data from 201 patients suggest that almost 70% had impairments in one or more organs four months after their initial symptoms of SARS-CoV-2 infection.
SARS-CoV-2 infection and persistence in the human body and brain at autopsy
We show that SARS-CoV-2 is widely distributed, predominantly among patients who died with severe COVID-19, and that virus replication is present in multiple respiratory and non-respiratory tissues, including the brain, early in infection. Further, we detected persistent SARS-CoV-2 RNA in multiple anatomic sites, including throughout the brain, as late as 230 days following symptom onset in one case. Despite extensive distribution of SARS-CoV-2 RNA throughout the body, we observed little evidence of inflammation or direct viral cytopathology outside the respiratory tract. Our data indicate that in some patients SARS-CoV-2 can cause systemic infection and persist in the body for months.
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Endothelial dysfunction in COVID-19: an overview of evidence, biomarkers, mechanisms and potential therapies

SARS-CoV-2 infection primarily affects the pulmonary system, but accumulating evidence suggests that it also affects the pan-vasculature in the extrapulmonary systems by directly (via virus infection) or indirectly (via cytokine storm), causing endothelial dysfunction (endotheliitis, endothelialitis and endotheliopathy) and multi-organ injury.
Mounting evidence suggests that SARS-CoV-2 infection leads to multiple instances of endothelial dysfunction, including reduced nitric oxide (NO) bioavailability, oxidative stress, endothelial injury, glycocalyx/barrier disruption, hyperpermeability, inflammation/leukocyte adhesion, senescence, endothelial-to-mesenchymal transition (EndoMT), hypercoagulability, thrombosis and many others. Thus, COVID-19 is deemed as a (micro)vascular and endothelial disease

Lingering cardiac involvement in previously well people after mild COVID-19

The mild but persistent non-ischemic cardiac inflammation that we describe in this study was not associated with overt structural heart disease or troponin release. Although it is triggered by a viral infection, profound myocardial injury or functional impairment is not typical, contrary to the classical definition of viral myocarditis2. Its pathophysiology is more reminiscent of findings in other chronic diffuse inflammatory syndromes that occur post-virally (for example, human immunodeficiency virus–associated cardiomyopathy)3or as a result of autoimmunity (for example, systemic lupus erythematosus4,5). In these cases, persistent subclinical cardiovascular inflammation seems to predispose people to a poor prognosis and the development of heart failure. Non-ischemic cardiac inflammatory involvement is therefore emerging as an important risk factor, and the long-term prognostic relevance of post-acute COVID-19 cardiac involvement in previously healthy people with mild initial COVID-19 illness requires further investigation.

​
July 27, 2020

Outcomes of Cardiovascular Magnetic Resonance Imaging in Patients Recently Recovered From Coronavirus Disease 2019 (COVID-19)

Conclusions and Relevance In this study of a cohort of German patients recently recovered from COVID-19 infection, CMR revealed cardiac involvement in 78 patients (78%) and ongoing myocardial inflammation in 60 patients (60%), independent of preexisting conditions, severity and overall course of the acute illness, and time from the original diagnosis. These findings indicate the need for ongoing investigation of the long-term cardiovascular consequences of COVID-19.
​

Post-COVID-19 Syndrome

Kamath, Vasantha; Anand, R.1; Radhakrishnan, Buvana1; Markanday, Kushal1
Post-COVID-19 “recovery” cannot be gauged solely on a negative polymerase chain reaction or hospital discharge. There is remarkable variation in the duration, severity, and fluctuation of symptoms, which can affect survivors’ quality of life, functional status, cognition, and mood, and lead to severe disability. Given the global scale of this pandemic, it is apparent that the health-care needs for patients with sequelae of COVID-19 will continue to increase for the foreseeable future.
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Immune system deviations?
We heard initial outbreaks of fungal infections of various types. Yes any infection can cause reduction in immune cells. Either because the immune system is busy diverted into tissues, fighting infection, or from apoptosis etc.
The deposits of antigens or viral pieces in many parts of the body, is perhaps causing problems, but also there appears to be markers of T cell exhaustion in some with high PD1 expression on some important immune cells too?
Long COVID following mild SARS-CoV-2 infection: characteristic T cell alterations and response to antihistamines Patients with long-COVID had reduced CD4+ and CD8+ effector memory (EM) cell numbers and increased PD-1 (programmed cell death protein 1) expression on central memory (CM) cells, whereas the asymptomatic participants had reduced CD8+ EM cells only and increased CD28 expression on CM cells. 72% of patients with long COVID who received HRA reported clinical improvement
The risk of pancreatic adenocarcinoma following SARS-CoV family infection
Our findings suggest that pancreatic adenocarcinoma is the most probably malignancy happening after infection with SARS-CoV family
The SARS-CoV-2 E protein induces Toll-like receptor 2-mediated neonatal lung injury in a model of COVID-19 viremia that is rescued by the glucocorticoid ciclesonide
This study provides insight into the pathogenesis of ALI and alveolar remodeling with SARS-CoV-2 viremia in children, whereas revealing the efficacy of steroids.
A new case-control study of Brazilian healthcare workers(HCWs) suggests as many as 27% developed long COVID after infection, and multiple infections raised the risk. The findings were published today in Infection Control & Hospital Epidemiology.
Estimates of the prevalence of long COVID, defined by the Centers for Disease Control and Prevention as new, returning, or lasting symptoms persisting 4 or more weeks after initial COVID-19 infection vary, with some studies showing as many as 43% of infected people will have some lingering symptoms 1 month after COVID-19 confirmation.
​
"39 (58%) patients (with glioma) experienced tumor progression following COVID-19 infection at a median of 34 days after testing positive for COVID-19.. 22 patients had measurably increased tumor area by a median of 63%; 18 of which constituted hyperprogression; 16 patients developed multifocal disease; 8 developed new nodular enhancement; 3 developed leptomeningeal disease (LMD); and 2 experienced increased infiltrative disease alone. 10 patients’ presentation with new glioma was preceded by COVID-19 infection by a median of 31 days. GBM (glioblastoma) patients represented the majority of progression events, among whom 59% progressed within 60 days of documented infection.. Glioma patients appear to have disease progression at an accelerated pace in the first two months after COVID-19 infection."
​
SARS-CoV-2 infection: The role of PD-1/PD-L1 and CTLA-4 axis
The most important diagnostic feature in individuals with COVID-19 is lymphocyte depletion, most importantly, T-cells. Due to the induction of interferon-γ (INF-γ) production by neutrophils and monocytes, which are abundantly present in the peripheral blood of the individuals with COVID-19, the expression of inhibitory immune checkpoints including, PD-1 (programmed death), PD-L1 and CTLA4 on the T-cells' surface is enhanced. The purpose of this review is to discuss the functions of these checkpoints and their effects on the dysfunction and exhaustion of T-cells, making them almost ineffective in individuals with COVID-19, especially in the cases with extreme symptoms.
​
COVID-19 and renal involvement: a prospective cohort study assessing the impact of mild SARS-CoV-2 infection on the kidney function of young healthy males
Mild COVID-19 is associated with mild renal involvement without AKI. Changes in the renal function appear to be related to reduced creatinine clearance and possible albumin leakage in the acute phase of the disease. The reduction in creatinine clearance is not predicted by viral load, and it appears to be a long-term effect of the disease that can last for at least 6 months.
​
Reduction and Functional Exhaustion of T Cells in Patients With Coronavirus Disease 2019 (COVID-19)
Conclusions: T cell counts are reduced significantly in COVID-19 patients, and the surviving T cells appear functionally exhausted. Non-ICU patients with total T cells counts lower than 800/μL may still require urgent intervention, even in the immediate absence of more severe symptoms due to a high risk for further deterioration in condition.
​
Brain
​
Cerebral blood flow in patients recovered from mild COVID-19
In this study, lower WM CBF, as well as widespread regional CBF changes identified using quantitative MRI, was found in mild COVID-19 patients. Further studies are needed to determine the reliability of this newly identified COVID-19 brain imaging marker and determine what drives these CBF changes.
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REVIEW

Alzheimer’s disease risk after COVID-19: a view from the perspective of the infectious hypothesis of neurodegeneration

Olivera, Eugenia; Sáez, Albany; Carniglia, Lila; Caruso, Carla; Lasaga, Mercedes; Durand, Daniela*
The present article aimed at reviewing the most recent literature supporting the infectious hypothesis of AD, and addressed the probable risk of developing Alzheimer-like dementia after severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, based on evidence linking viral infections and amyloidosis.
​

• EWS
• 10 February 2022

Heart-disease risk soars after COVID — even with a mild case

Massive study shows a long-term, substantial rise in risk of cardiovascular disease, including heart attack and stroke, after a SARS-CoV-2 infection.
JOURNAL ARTICLE

Global Prevalence of Post-Coronavirus Disease 2019 (COVID-19) Condition or Long COVID: A Meta-Analysis and Systematic Review

This study finds post-COVID-19 condition prevalence is substantial; the health effects of COVID-19 seem to be prolonged and can exert stress on the healthcare system.
Review articleBrain disorders: Impact of mild SARS-CoV-2 may shrink several parts of the brain
Coronavirus (COVID-19) is a highly infectious respiratory infection discovered in Wuhan, China, in December 2019. As a result of the pandemic, several individuals have experienced life-threatening diseases, the loss of loved ones, lockdowns, isolation, an increase in unemployment, and household conflict. Moreover, COVID-19 may cause direct brain injury via encephalopathy. The long-term impacts of this virus on mental healthand brain function need to be analysed by researchers in the coming years. This article aims to describe the prolonged neurological clinical consequences related to brain changes in people with mild COVID-19 infection. When compared to a control group, people those who tested positive for COVID-19 had more brain shrinkage, grey matter shrinkage, and tissue damage. The damage occurs predominantly in areas of the brain that are associated with odour, ambiguity, strokes, reduced attention, headaches, sensory abnormalities, depression, and mental abilities for few months after the first infection. Therefore, in patients after a severe clinical condition of COVID-19, a deepening of persistent neurological signs is necessary.
​
continuing ...

Global Info Research announces the release of the report “Global Halogen Free Polyimide Coverlay Market 2023 by Manufacturers, Regions, Type and Application, Forecast to 2029” . The report is a detailed and comprehensive analysis presented by region and country, type and application. As the market is constantly changing, the report explores the competition, supply and demand trends, as well as key factors that contribute to its changing demands across many markets. Company profiles and product examples of selected competitors, along with market share estimates of some of the selected leaders for the year 2023, are provided. In addition, the report provides key insights about market drivers, restraints, opportunities, new product launches or approvals, COVID-19 and Russia-Ukraine War Influence.
​
According to our (Global Info Research) latest study, the global Halogen Free Polyimide Coverlay market size was valued at USD million in 2022 and is forecast to a readjusted size of USD million by 2029 with a CAGR of % during review period. The influence of COVID-19 and the Russia-Ukraine War were considered while estimating market sizes.
​
Key Features:
Global Halogen Free Polyimide Coverlay market size and forecasts, in consumption value), sales quantity, and average selling prices, 2018-2029
Global Halogen Free Polyimide Coverlay market size and forecasts by region and country, in consumption value, sales quantity, and average selling prices, 2018-2029
Global Halogen Free Polyimide Coverlay market size and forecasts, by Type and by Application, in consumption value, sales quantity, and average selling prices, 2018-2029
Global Halogen Free Polyimide Coverlay market shares of main players, shipments in revenue, sales quantity, and ASP, 2018-2023
​
The Primary Objectives in This Report Are:
To determine the size of the total market opportunity of global and key countries
To assess the growth potential for Halogen Free Polyimide Coverlay
To forecast future growth in each product and end-use market
To assess competitive factors affecting the marketplace
This report profiles key players in the global Halogen Free Polyimide Coverlay market based on the following parameters - company overview, production, value, price, gross margin, product portfolio, geographical presence, and key developments.
​
Request Free Sample Copy Or buy this report at:
https://www.globalinforesearch.com/reports/1535535/halogen-free-polyimide-coverlay
The Halogen Free Polyimide Coverlay market is segmented as below:
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Market segment by Type
Black
White
Yellow
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Market segment by Application
Smartphone
Laptop
LCD
HDD
Other
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Major players covered
TAIFLEX
Hubei Omar Electronics Technology
Asia Electronic Materials
Ventec
Arisawa
DuPont
SYTECH
Hanhwa
NIKKAN
Dongyi
Anhui Xinchen Optical New Material
INNOX Advanced Materials
​
The Main Contents of the Report, includes a total of 15 chapters:
Chapter 1, to describe Halogen Free Polyimide Coverlay product scope, market overview, market estimation caveats and base year.
Chapter 2, to profile the top manufacturers of Halogen Free Polyimide Coverlay, with price, sales, revenue and global market share of Halogen Free Polyimide Coverlay from 2018 to 2023.
Chapter 3, the Halogen Free Polyimide Coverlay competitive situation, sales quantity, revenue and global market share of top manufacturers are analyzed emphatically by landscape contrast.
Chapter 4, the Halogen Free Polyimide Coverlay breakdown data are shown at the regional level, to show the sales quantity, consumption value and growth by regions, from 2018 to 2029.
Chapter 5 and 6, to segment the sales by Type and application, with sales market share and growth rate by type, application, from 2018 to 2029.
Chapter 7, 8, 9, 10 and 11, to break the sales data at the country level, with sales quantity, consumption value and market share for key countries in the world, from 2017 to 2022.and Halogen Free Polyimide Coverlay market forecast, by regions, type and application, with sales and revenue, from 2024 to 2029.
Chapter 12, market dynamics, drivers, restraints, trends, Porters Five Forces analysis, and Influence of COVID-19 and Russia-Ukraine War.
Chapter 13, the key raw materials and key suppliers, and industry chain of Halogen Free Polyimide Coverlay.
Chapter 14 and 15, to describe Halogen Free Polyimide Coverlay sales channel, distributors, customers, research findings and conclusion.
​
The analyst presents a detailed picture of the market by the way of study, synthesis, and summation of data from multiple sources by an analysis of key parameters. Our report on the Halogen Free Polyimide Coverlay market covers the following areas:
1.Halogen Free Polyimide Coverlay market sizing
2.Halogen Free Polyimide Coverlay market forecast
3.Halogen Free Polyimide Coverlay market industry analysis
4.Analyze the needs of the global Halogen Free Polyimide Coverlay business market
5.Answer the market level of global Halogen Free Polyimide Coverlay
6.Statistics the annual growth of the global Halogen Free Polyimide Coverlay production market
7.The main producers of the global Halogen Free Polyimide Coverlay production market
8.Describe the growth factor that promotes market demand
​
Global Info Research is a company that digs deep into global industry information to support enterprises with market strategies and in-depth market development analysis reports. We provides market information consulting services in the global region to support enterprise strategic planning and official information reporting, and focuses on customized research, management consulting, IPO consulting, industry chain research, database and top industry services. At the same time, Global Info Research is also a report publisher, a customer and an interest-based suppliers, and is trusted by more than 30,000 companies around the world. We will always carry out all aspects of our business with excellent expertise and experience.
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Contact Us:
Global Info Research
Web: https://www.globalinforesearch.com
CN: 0086-176 6505 2062
HK: 00852-5819 7708
US: 001-347 966 1888
Email: [email protected]
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New job, not been paid on time.
Hi all, this is my first post so be kind!
I am a barmaid in a tiny village pub in the UK. I’m 22 next week. I started a new job on the 16th May so coming up to a month now. On a number of occasions in the first week I asked my manager if there was a contract for me to sign, and any paperwork to fill out. She told me her printer was broken and so, eager to be ahead of everything, I said I’d print it myself and bring it in if it was going to be an issue. Within the next couple of days, I brought the paperwork in, all filled out and ready to be added to payroll. Speaking to a couple of the girls I work with, they all told me that pay day is 7th of every month..strange day but okay, fine.
After being told that, I changed a lot of my direct debits and delayed some payments until this date so I can be on time and avoid fines etc.
However, 6th June comes around and we were informed on the work group chat that manager was ill and told a lot of the waitresses not to come into work as food wasn’t on for the next couple of days.
All day 7th we were waiting for pay to hit. It got to 12:10am last night (8th) and a couple girls started making comments in the group chat about needing to be paid. At 3am this morning, she posted this in the group chat… ‘Everyone except Alice and Hannah has been paid. Can’t do them from bed as they need to be set up as new. Will do as soon as I’m well’
I understand that the manager is ill but it’s not my problem. I gave her my documents and account details with more than enough time to be added, to be paid on the same day as everyone else.
She’s cancelled food for the whole weekend so intends on being off until start of next week.
Is there anything I can do, or politely say to her about this? I don’t have enough fuel to get to work today-Sunday without being paid.
What rights do I have in terms of not showing up? I didn’t sign any contracts, so where do I stand?

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Have big projects that need to be done ASAP? You can count on me!
Looking for long-term gigs, but one-time writing projects would be perfect as well!!!
My rate starts at $0.10 per word but can be negotiable depending on the type and amount of writing work to be done. The payment method is through Paypal, Payoneer, or Wise.
Interested in SEO services? Let's hop on a call and see how I can be of help. I'm ready to show you some of my successful SEO case studies as well!
Shoot me a message here on Reddit or feel free to contact me at [email protected]

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